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<section><h2 id="relatedArticles" class="top">Related Articles</h2><div class="section-paragraph linked-article"><ul class="rlist"><li class="linked-article__item"><a href="../article/S0735-6757(15)01042-6/fulltext" title="Bilateral facial nerve paralysis in the outpatient setting; the need for follow-up in the face of a serious pathology" class="linked-article__title link--underlined">Bilateral facial nerve paralysis in the outpatient setting; the need for follow-up in the face of a serious pathology</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 34</span><span>Issue 7</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-0" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-0" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>Bilateral facial nerve paralysis is an extremely rare disease with an annual incidence of 1 in 5 million cases. Although unilateral facial nerve paralysis is often a benign illness, bilateral facial nerve palsy is a harbinger of more serious pathology. We present the case of a 25-year-old man who presented with alternating left then right facial weakness. We diagnosed him with bilateral facial nerve palsy and scheduled further diagnostic tests, but he was lost to follow-up. Given the severity of the differential diagnosis of bilateral facial nerve paralysis, diagnostic tests must be done at time of initial presentation.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/S0735-6757(15)01042-6/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="../article/S0735-6757(15)01042-6/pdf" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="../article/S0735-6757(18)30316-4/fulltext" title="Facial nerve palsy following mild mastoid trauma on trampoline" class="linked-article__title link--underlined">Facial nerve palsy following mild mastoid trauma on trampoline</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 36</span><span>Issue 8</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-1" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-1" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>We present a case report of a 14-year-old boy who was jumping on a trampoline when he struck his right mastoid on a support pole. The following day, he developed a right-sided facial droop and inability to close his right eye. He presented to the emergency department, where CT of his temporal bone was negative and he was started on prednisone. Over the next month, he had spontaneous recovery of his facial nerve (FN) function.In cases of traumatic FN palsy, urgent referral to otolaryngology is needed, even without a fracture of the temporal bone, as edema within the facial nerve could require decompressive surgery.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/S0735-6757(18)30316-4/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="/" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="/" title="Bilateral facial paralysis in a non-Hodgkin lymphoma during remission period: a rare but an important condition to be investigated" class="linked-article__title link--underlined">Bilateral facial paralysis in a non-Hodgkin lymphoma during remission period: a rare but an important condition to be investigated</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 34</span><span>Issue 12</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-2" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-2" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>Lymphomatous meningitis (LM) is a complication of systemic non-Hodgkin lymphomas. Clinical features include confusion, seizures, headaches, and cranial nerve (CN) palsies. The facial nerve is the CN usually affected; however, bilateral palsy is considered rare in LM. We present the case of a 62-year-old man who showed lower extremity weakness and bilateral facial paralysis chiefly involving the upper lip, with inability to smile and whistle. His medical history was significant for stage IV diffuse large B-cell lymphoma which had been treated with chemotherapy some months ago.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="/" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="../article/S0735-6757(16)30232-7/pdf" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="/" title="Facial nerve palsy in a 3-year-old child with severe hypertension" class="linked-article__title link--underlined">Facial nerve palsy in a 3-year-old child with severe hypertension</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 33</span><span>Issue 12</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-3" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-3" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>We report an interesting case of a child with new-onset malignant hypertension (HTN) associated with facial paralysis. A review of the medical literature on this association and discussion of diagnostic and management aspects are included.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="/" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="../article/S0735-6757(15)00324-1/pdf" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="../article/S0735-6757(07)00473-1/fulltext" title="Mastoid bone fracture presenting as unusual delayed onset of facial nerve palsy" class="linked-article__title link--underlined">Mastoid bone fracture presenting as unusual delayed onset of facial nerve palsy</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 26</span><span>Issue 3</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-4" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-4" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>Delayed-onset facial nerve paralysis is a rather uncommon complication of a mastoid bone fracture for children younger than 10 years. We routinely arrange a cranial computed tomography (CT) for patients encountering initial loss of consciousness, severe headache, intractable vomiting, and/or any neurologic deficit arising from trauma to the head. However, minor symptomatic cranial nerve damage may be missed and the presenting symptom diagnosed as being a peripheral nerve problem. Herein, we report a case of a young boy who presented at our emergency department (ED) 3 days subsequent to his accident, complaining of hearing loss in the right ear and paralysis of the ipsilateral face.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/S0735-6757(07)00473-1/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="../article/S0735-6757(07)00473-1/pdf" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="../article/S0735-6757(20)30313-2/fulltext" title="An emergency medicine-focused review of malignant otitis externa" class="linked-article__title link--underlined">An emergency medicine-focused review of malignant otitis externa</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-5" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-5" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>Malignant otitis externa (MOE) is a progressive infection of the external auditory canal (EAC). This disease is rare but has severe morbidity and mortality.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/S0735-6757(20)30313-2/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="/" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="../article/0735-6757(95)90096-9/fulltext" title="Temporal bone fractures" class="linked-article__title link--underlined">Temporal bone fractures</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 13</span><span>Issue 2</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-6" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-6" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>The diagnosis and monitoring of patients presenting to an emergency department with blunt temporal bone fracture and complications requiring acute management were reviewed for a four-month period. Of 104 trauma patients with closed head injury, 15 patients were diagnosed with temporal bone fracture, 12 of whom survived their injuries. Four patients developed cerebrospinal fluid (CSF) otorrhea and two patients developed facial nerve paralysis; all patients had resolution of complications with conservative management.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/0735-6757(95)90096-9/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="../article/0735-6757(95)90096-9/pdf" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="../article/0735-6757(89)90051-X/fulltext" title="Emergency presentation of coalescent mastoiditis" class="linked-article__title link--underlined">Emergency presentation of coalescent mastoiditis</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 7</span><span>Issue 4</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-7" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-7" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>Since the introduction of sulfonamides in the late 1930s, coalescent mastoiditis is an uncommon complication of otitis media. Swelling, erythema, and tenderness over the mastoid area with associated anterior displacement of the auricle are classic signs and symptoms of coalescent mastoiditis with formation of a subperiosteal abscess. Early recognition of this entity is essential to prevent its complications. Prompt otolaryngologic consultation is indicated and inpatient antibiotic therapy should be begun.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/0735-6757(89)90051-X/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="/" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="../article/S0735-6757(16)30442-9/fulltext" title="Multiple cranial nerve neuropathies, ataxia and, areflexia: Miller Fisher syndrome in a child and review" class="linked-article__title link--underlined">Multiple cranial nerve neuropathies, ataxia and, areflexia: Miller Fisher syndrome in a child and review</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 35</span><span>Issue 4</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-8" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-8" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>Ataxia in children mainly occurs from postinfectious cerebellar ataxia, toxic ingestions, and labrynthitis. An unusual cause is the Miller Fisher syndrome (MFS) which is considered a variant of Guillain-Barré syndrome. It is a self-limiting demyelinating disease of the peripheral nervous system characterized by the triad of ophthalmoplegia, cerebellar ataxia, and areflexia. We report on MFS in a child with review of the medical literature. The medical literature was searched using MEDLINE/PubMed, Cochrane Library, and EMBASE.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="../article/S0735-6757(16)30442-9/fulltext" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="../article/S0735-6757(16)30442-9/pdf" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li><li class="linked-article__item"><a href="/" title="Acute transient hemiparesis induced by lightning strike" class="linked-article__title link--underlined">Acute transient hemiparesis induced by lightning strike</a><div class="linked-article__labels"></div><div class="linked-article__details"><span><i>The American Journal of Emergency Medicine</i></span><span>Vol. 33</span><span>Issue 7</span></div><ul class="rlist rlist--inline linked-article__links clearfix"><li class="bulleted preview-label"><a href="#" data-db-target-for="related-article-in-brief-9" class="link--underlined">In Brief</a><div data-db-target-of="related-article-in-brief-9" class="dropBlock__holder__article-preview dropBlock__holder"><div class="dropBlock__body"><span>According to data from the National Oceanic and Atmospheric Administration, in the years from 1959 to 1994, lightning was responsible for more than 3000 deaths and nearly 10,000 casualties. The most important characteristic features of lightning injuries are multisystem involvement and widely variable severity. Lightning strikes are primarily a neurologic injury that affects all 3 components of the nervous system: central, autonomic, and peripheral. Neurologic complications of lightning strikes vary from transient benign symptoms to permanent disability.</span></div><a href="#" title="Close info" class="dropBlock__ctrl--close"><i aria-hidden="true" class="icon-close_thin"></i></a></div></li><li class="bulleted"><a href="/" title="Full-Text" class="faded link--underlined">Full-Text</a></li><li><a href="/" title="PDF" target="_blank" class="link--underlined">PDF</a></li></ul></li></ul></div></section>

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